VOL. 7, No. 1/2, December 2011

J. Stuger [2011] Med. Hypotheses Res. 7: 57-69.

Cancer is the Reactivation of Abort Mechanisms from
Embryogenesis in Later Stages of Life

Jerry Stuger*

Overvoorde 19, 1082 GA, Amsterdam, The Netherlands

Abstract. Cancer is the reactivation of mechanisms from embryogenesis which were
originally used for detecting defects or malfunctions in the developing fetus by spontaneously
aborting fetuses with chromosomal abnormalities. Cancer remobilizes and reactivates those
mechanisms to fulfill its specific purpose which is the termination of the organism. This is in
my opinion the genuine purpose for the ‘growth till death of the organism’ process as seen in
cancer. The cancer process starts with the altering of the status of a distressed ‘detection cell’
from mature to immature which activates the uncontrolled growth mechanism. It is suspected
that this cell signaling alters specific embryogenic genes for this purpose.
The most important feature is the blocking or disrupting of the highly regulated intracellular
control processes. Because of the disruption of the internal cell cycle mechanism the
distressed ‘detection cell’ will continue only as a growth and proliferating machine which is
facilitated by mechanisms which support and stimulate this goal. These are the so called
cancer superpowers. These detection cells are the initiators of the cancer process. A network
of these special kinds of cells monitor when an organ or tissue has reached its mature form
and size. It is assumed they also monitor the homeostasis in the cellular micro environment
of the constituent parts of the organism.
Another example of the use of mechanisms from embryogenesis is metastases which show
striking similarities with the mechanisms used in the fertilization process by sperm cells. It is
suspected that the mechanisms of metastases are triggered by expressing embryonic sperm
genetic complements in specific cancer cells.

* Correspondence: Dr. Jerry Stuger, Overvoorde 19, 1082 GA, Amsterdam, The Netherlands.